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The bradykinin hypothesis also accounts for Covid-19's neurological effects, by Hamz
The bradykinin hypothesis also accounts for Covid-19's neurological effects, by Hamz
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SARS-CoV-2 and the spike protein in endotheliopathy: Trends in Microbiology
The neuropsychiatric manifestations of COVID-19: Interactions with psychiatric illness and pharmacological treatment - ScienceDirect
JPM, Free Full-Text
Neurological complications and infection mechanism of SARS-CoV-2
Biomedicines, Free Full-Text
Cells, Free Full-Text
Bradykinin Activates the Bradykinin B2 Receptor to Ameliorate Neuronal Injury in a Rat Model of Spinal Cord Ischemia-Reperfusion Injury
Bradykinin Hypothesis - Most Compelling Explanation of COVID-19 Yet
Clinical Implications of SARS-CoV-2 Interaction With Renin Angiotensin System: JACC Review Topic of the Week
ACE2 expression in rat brain: Implications for COVID-19 associated neurological manifestations - ScienceDirect
The mechanism underlying extrapulmonary complications of the coronavirus disease 2019 and its therapeutic implication
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